Welcome to my talk page! Nphilip (talk) 14:07, 11 October 2013 (UTC)NphilipNphilip (talk) 14:07, 11 October 2013 (UTC)
Peer Review
I found the page both readable and informative, a rare mixture in Neuroscience literature (at least in my experience). You cite sources consistency, adding a tone of authority to your writing, and demonstrate clear mastery of neuroscience concepts, connecting physiological and mental symptoms to brain anatomy in clear prose. For example, The paragraph:
The atrophy is progressive; early symptoms include difficulty reading, blurred vision, light sensitivity, issues with depth perception, and trouble navigating through space.[7][8] Additional symptoms include Apraxia, a movement disorder, Alexia, a reading disorder, and Visual agnosia, an object recognition disorder.[9] Damage to the ventral, or “what” stream, of the visual system in the temporal lobe leads to the symptoms related to general vision and object recognition deficits, and damage to the dorsal, or “where/how” stream in the parietal lobe, leads to PCA symptoms related to impaired movements in response to visual stimuli, such as navigation and Apraxia.[9][10]"
Defines all concepts, making it understandable for those unfamiliar to neuroscience but also doesn't oversimplify concepts. Sure, someone who has not taken a neuroscience course might struggle a bit with the text. But ultimately, they would have a clear, if somewhat impomplete understanding.
Another thing I like is that you integrated its connection to Alzheimer's disease throughout the article. This may seem like a trivial point. However, Alzheimer's disease is a household name whereas Posterior Cortical Atrophy is not. Making the connection clear throughout helps contextualize this disorder and provide practical information for someone who suffers or knows someone who suffers from this condition and would need to know how to treat it differently.
An area that reads less clearly however is:
"Magnetic Resonance Imaging (MRI's) taken of a PCA patient during the early stages of the disease can show brain atrophy in the right posterior lobe and occipital gyrus. PCA imaging can contradict the common MRI images of AD patients, which shows atrophy in the medial temporal cortex.[14][17] Single-photon emission computed tomography and Positron emission tomography studies can also be used."
You discard the technique used in the former paragraph I pulled and do not define the neuroscience concepts rendering them meaningless for most people. David Ullmann 1994 (talk) 18:16, 26 November 2013 (UTC) — Preceding unsigned comment added by David Ullmann 1994 (talk • contribs) 03:41, 26 November 2013 (UTC)