User:RedBeeV/sandbox

Candidates

https://en.wikipedia.org/wiki/Neuroinflammation

The page for neuroinflammation points out it's correlation with neurodegenrative diseases, but they're not the only correlated diseases, so that's what this new section is about.

Relationship with other diseases edit

(1st draft)

Sources

http://cornell.summon.serialssolutions.com/search?s.fvf=ContentType,Newspaper+Article,t&s.q=inflammation%20in%20individuals%20with%20schizophrenia#!/search?ho=t&fvf=ContentType,Newspaper%20Article,t&l=en&q=inflammation%20in%20individuals%20with%20schizophrenia

Neuroinflammation has also been found to be heavily correlated with mental illnesses such as schizophrenia and bipolar disorder. In the immunologic theory of schizophrenia, neuroinflammation is one of the main causes of the disease. Neuroinflammation is also associated with other nondegenrative mental illnesses such as depression, bipolar disorder, and anxiety. One hypothesis is that prenatal infections that cause inflammation of the CNS disrupts normal growth behavior. ^[1] The neuropathogenetic theory suggests that neuroprogression to schizophrenia begins through flawed early cell programming that disrupts the blood brain barrier so that neuroinflammation occurs.^[2] Multiple studies link inflammation and these diseases.^[3]^[4] There have also been studies showing the expression of neuroinflammation genetically.^[5]

Neuroinflammation

References edit

1. https://www-sciencedirect-com.proxy.library.cornell.edu/science/article/pii/S0149763413002753

This is a user sandbox of RedBeeV. You can use it for testing or practicing edits.
This is not the sandbox where you should draft your assigned article for a dashboard.wikiedu.org course.
To find the right sandbox for your assignment, visit your Dashboard course page and follow the Sandbox Draft link for your assigned article in the My Articles section.

Get Help

^ Feigenson, Keith A.; Kusnecov, Alex W.; Silverstein, Steven M. (2014-01). "Inflammation and the two-hit hypothesis of schizophrenia". Neuroscience & Biobehavioral Reviews. 38: 72–93. doi:10.1016/j.neubiorev.2013.11.006. ISSN 0149-7634. PMC 3896922. PMID 24247023. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)
^ Berk, Michael; Debnath, Monojit (2014-11-01). "Th17 Pathway–Mediated Immunopathogenesis of Schizophrenia: Mechanisms and Implications". Schizophrenia Bulletin. 40 (6): 1412–1421. doi:10.1093/schbul/sbu049. ISSN 0586-7614. PMC 4193719. PMID 24711545.{{cite journal}}: CS1 maint: PMC format (link)
^ Weickert, C. Shannon; Webster, M. J.; Fung, S. J.; Sinclair, D.; Fillman, S. G. (2014-02). "Markers of inflammation and stress distinguish subsets of individuals with schizophrenia and bipolar disorder". Translational Psychiatry. 4 (2): e365. doi:10.1038/tp.2014.8. ISSN 2158-3188. PMC 3944638. PMID 24569695. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)
^ www.tandfonline.com. doi:10.1586/14737175.7.7.789 https://www.tandfonline.com/action/captchaChallenge?redirectUri=%2Fdoi%2Ffull%2F10.1586%2F14737175.7.7.789&. Retrieved 2018-12-17. {{cite web}}: Missing or empty |title= (help)
^ Kim, S.; Lee, D.; Webster, M. J.; Seo, J.-S.; Kim, J.-Ii; Shin, J.-Y.; Kim, J.; Hwang, Y. (2013-10). "Gene expression profiling by mRNA sequencing reveals increased expression of immune/inflammation-related genes in the hippocampus of individuals with schizophrenia". Translational Psychiatry. 3 (10): e321. doi:10.1038/tp.2013.94. ISSN 2158-3188. PMC 3818014. PMID 24169640. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)

[1] Feigenson, Keith A.; Kusnecov, Alex W.; Silverstein, Steven M. (2014-01). "Inflammation and the two-hit hypothesis of schizophrenia". Neuroscience & Biobehavioral Reviews. 38: 72–93. doi:10.1016/j.neubiorev.2013.11.006. ISSN 0149-7634. PMC 3896922. PMID 24247023. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)

[2] Berk, Michael; Debnath, Monojit (2014-11-01). "Th17 Pathway–Mediated Immunopathogenesis of Schizophrenia: Mechanisms and Implications". Schizophrenia Bulletin. 40 (6): 1412–1421. doi:10.1093/schbul/sbu049. ISSN 0586-7614. PMC 4193719. PMID 24711545.{{cite journal}}: CS1 maint: PMC format (link)

[3] Weickert, C. Shannon; Webster, M. J.; Fung, S. J.; Sinclair, D.; Fillman, S. G. (2014-02). "Markers of inflammation and stress distinguish subsets of individuals with schizophrenia and bipolar disorder". Translational Psychiatry. 4 (2): e365. doi:10.1038/tp.2014.8. ISSN 2158-3188. PMC 3944638. PMID 24569695. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)

[4] www.tandfonline.com. doi:10.1586/14737175.7.7.789 https://www.tandfonline.com/action/captchaChallenge?redirectUri=%2Fdoi%2Ffull%2F10.1586%2F14737175.7.7.789&. Retrieved 2018-12-17. {{cite web}}: Missing or empty |title= (help)

[5] Kim, S.; Lee, D.; Webster, M. J.; Seo, J.-S.; Kim, J.-Ii; Shin, J.-Y.; Kim, J.; Hwang, Y. (2013-10). "Gene expression profiling by mRNA sequencing reveals increased expression of immune/inflammation-related genes in the hippocampus of individuals with schizophrenia". Translational Psychiatry. 3 (10): e321. doi:10.1038/tp.2013.94. ISSN 2158-3188. PMC 3818014. PMID 24169640. {{cite journal}}: Check date values in: |date= (help)CS1 maint: PMC format (link)

[1]

[2]

[3]

[4]

[5]